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human umbilical vein endothelial cells huvecs  (ATCC)


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    ATCC human umbilical vein endothelial cells huvecs
    Human Umbilical Vein Endothelial Cells Huvecs, supplied by ATCC, used in various techniques. Bioz Stars score: 99/100, based on 4978 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/primary+human+umbilical+vein+endothelial+cells+huvecs/Primary+Umbilical+Vein+Endothelial+Cells%3B+Normal%2C+Human/pmc13676988-271-0-17
    Average 99 stars, based on 4978 article reviews
    human umbilical vein endothelial cells huvecs - by Bioz Stars, 2026-10
    99/100 stars

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    Cell Culture:

    Article Title: Geranylgeranyl transferase inhibitors mitigate intestinal radiation toxicity.
    Article Snippet: Please cite this article as: Katherine McGinness BS , Qudes Al-anbaky PhD , Enoch K. Larrey MS , Mitchell Rotenberry BS , Harrison Cook BS , Edith Nathalie Pineda MS , Ruofei Du PhD , Marjan Boerma PhD , Rupak Pathak PhD , Geranylgeranyl transferase inhibitors mitigate intestinal radiation toxicity, International Journal of Radiation Oncology, Biology, Physics (2025), doi: https://doi.org/10.1016/j.ijrobp.2025.10.026

    Article Title: Extracellular Vesicles Derived from VEGF mRNA-Engineered Mesenchymal Stem Cells Promote Endothelial Cell Survival
    Article Snippet: .. Primary human umbilical vein endothelial cells (HUVECs) (ATCC, Manassas, VA, USA, catalog# PCS-100-010) were cultured in HUVEC growth medium composed of Vascular Cell Basal Medium (ATCC, catalog# PCS-100-030) supplemented with the Endothelial Cell Growth Kit-VEGF (ATCC, catalog# PCS-100-014) and 0.1% Penicillin–Streptomycin solution (Gibco, catalog# 15140122). ..

    Article Title: CEP78 regulates the proliferation and angiogenesis of endothelial cells by mediating microtubule stabilization via p150glued.
    Article Snippet: Angiogenesis, which is defined as the sprouting of nascent vasculature from established endothelial networks, constitutes a vital biological mechanism underpinning embryonic morphogenesis and wound regeneration processes (Shaw et al. 2024).. This process is tightly regulated by a variety of signaling molecules, such as vascular endothelial growth factor (VEGF) and fibroblast growth factor (FGF), which control endothelial cell proliferation, migration, and tube formation (Shaw et al. 2024; Zeng and Fu 2025).. Dysregulation of angiogenesis can lead to numerous pathological conditions, including cancer, diabetic retinopathy and cardiovascular diseases (Kishishita et al. 2024; Yang et al. 2024; Zhang 2024).

    Article Title: Lysolecithin reprogramming via LPCAT1 modulation restores endothelial function and prevents diabetes-associated dysmetabolism
    Article Snippet: .. Primary human umbilical vein endothelial cells (HUVECs) were obtained from American Type Culture Collection (ATCC; PCS-100-010) and cultured in Vascular Cell Basal Medium (ATCC; PCS-100-030) supplemented with Endothelial Cell Growth Kit (ATCC; PCS-110-041), penicillin (100 U/ml), and streptomycin (100 U/ml) (ATCC; Cat# 30–2300). ..

    Article Title: Impact of pancreatic proenzymes on pancreatic ductal adenocarcinoma associated fibroblasts.
    Article Snippet: .. Primary human umbilical vein endothelial cells (HUVECs) were also obtained from the ATCC (PCS-100-010) and cultured in EGM-2 growth medium (Lonza, Basel, Switzerland) supplemented with 2% fetal bovine serum (FBS) and endothelial growth supplement (BD Biosciences, San Diego, CA). ..

    Article Title: Impact of pancreatic proenzymes on pancreatic ductal adenocarcinoma associated fibroblasts
    Article Snippet: .. Primary human umbilical vein endothelial cells (HUVECs) were also obtained from the ATCC (PCS-100-010) and cultured in EGM-2 growth medium (Lonza, Basel, Switzerland) supplemented with 2% fetal bovine serum (FBS) and endothelial growth supplement (BD Biosciences, San Diego, CA). ..

    Isolation:

    Article Title: SLC22A6-dependent lactylation of H3K9 aggravates endothelial dysfunction and atherosclerosis.
    Article Snippet: Background: Atherosclerosis, a leading cause of cardiovascular morbidity and mortality, is driven by endothelial dysfunction.. While metabolic reprogramming toward glycolysis in endothelial cells exacerbates disease progression, the role of lactate-derived lactylation in atherogenesis remains poorly understood.. Methods: We performed RNA-seq on aortic tissues from atherosclerotic mice to identify differentially expressed genes, along with Seahorse XF metabolic flux analysis.



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